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Oxidative Stress and Blood–Brain Barrier Dysfunction Under Particular Consideration of Matrix Metalloproteinases

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An experimental study performed short-term and long-term lutein administration to type 1 diabetic rats and found that lutein inhibited N-epsilon-(carboxymethyl)lysine and N-epsilon-(carboxyethyl)lysine and inhibited the progression of cataractogenesis in the lens of diabetic rats [63]