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(2011) reported that pretreatment of A549 cells with synthetic antioxidant N -acetylcysteine enhanced GSH levels in A549 cells, however, the mechanism that underlies this increase in intracellular GSH, and whether this involved enhanced GRed enzymatic activity was not explored

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Therefore, targeting PRMT1 holds promise as a novel therapeutic strategy, particularly in kidney stone disease, in which metabolic derangement and inflammation are prevalent

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