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The Δ gshT mutant, which lacks the gene that encodes for a GSH transporter [9], confirmed that the intracellular availability of GSH is needed for a typical OSR in S

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Krivic et al., 2008) ligament-healing (Cerovecki et al., 2010) and has a strong osteogenic effect the in non-union model (Sebecic et al., 1999) (and in other models, such as is observed with rat femoral head osteonecrosis (Gamulin et al., 2013), or alveolar bone loss (Keremi et al., 2009)