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Our results provide a possible mechanism in which upregulation of phosphorylated p53 and p16 maintain the cell cycle arrest of keloid fibroblasts, and FOXO4-DRI selectively induces apoptosis of keloid senescent fibroblasts by prompting p53-pS15 nuclear exclusion

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NNMT is overexpressed in several tumor types including gastric and thyroid cancers, and inhibiting it has been proposed as anti-tumor in those contexts