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The double staining of human Tau (HT7) and Ser422‐phosphorylated Tau (pS422) showed a marked decrease in the number of pS422‐positive neurons, a pathological marker of severe Tau, and Gallyas‐positive NFTs after treating Tau transgenic mice with selenate

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(2012), neither short-term (5 min) nor long-term (2 weeks) administration of NAC resulted in the elevation of GSH levels in the healthy organs of rats [25]